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Given that immune cells, especially immune cells, can release IL-1β and prostaglandin E 2 (PGE2) further studies show that Glyergic synapses exposed to IL-1β exposure induces LTP in glycinergic synapses, while PGE2 selectively inhibits excitatory signals of glycinergic synapses on non-glycinergic neurons, thereby suppressing glycinergic inhibition and promoting chronic pain (185, 186)

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We measure, we understand, and we correct the underlying chemistry