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Proposed pharmacology (mostly animal data) • Promotes angiogenesis via VEGFR2-Akt-eNOS signaling • Boosts nitric oxide through the Src-Caveolin-1-eNOS pathway • Upregulates growth hormone receptors on tendon fibroblasts • Lowers pro-inflammatory cytokines (TNF-alpha, IL-6) No specific high-affinity receptor has ever been identified

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doi: 10.1016/S0026-895X(24)26516-0