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Previous studies reported that hepatocellular damage in COVID-19 patients was likely induced by medications, a systemic inflammatory response, and the sequelae of hypoxia-ischemia reperfusion injury ( While existing ML approaches for predicting hepatic toxicity have provided important mechanistic insights through modeling, significant translational limitations persist ( Overall, our AI-driven framework facilitated timely risk stratification by performing continuous multivariate pattern analysis of electronic health records (EHRs), thereby triggering protocolized confirmatory retesting and reducing diagnostic delays in the detection of subclinical hepatotoxicity
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