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There is accumulating evidence from experimental animal and tissue culture studies that full radical‐mediated oxidative stress is implicated in the pathogenesis of diabetic polyneuropathy by inducing neurovascular defects that result in endoneurial hypoxia and subsequent nerve dysfunction (Kellogg & Pop‐Busui, 2005)

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Variants:Bloom B2G1 (3pcs)

1998;332(Part 1):251–5